Whodunnit? {unabridged}

Written by Martin Neil , Jessica Hockett , Jonathan Engler , and Norman Fenton on September 4, 2023. Posted in Current News

Was SARS-CoV-2 or Pneumonia the primary cause of respiratory Covid-19 deaths?

We have investigated the pneumonia hypothesis: that a proportion of covid-19 deaths, those with associated respiratory symptoms (rather than deaths coded as covid-19 because of a positive PCR test, that are absent symptoms), were caused by bacterial pneumonia, and that bacterial pneumonia was the primary, not the secondary, infection.

Our argument in favour of the hypothesis is:

• Conflating pneumonia & covid-19 repeats an official longstanding tactic of conflating the attribution of influenza and pneumonia. The reduction in the public’s perceived threat of flu may have prompted the pharmaceutical industry to attempt a rebranding of the threat along with a new suite of marketable products to respond to that threat.
• We investigated the hypothesis that a proportion of covid-19 deaths, those with associated respiratory symptoms (rather than deaths coded as covid-19 because of a positive PCR test, that are absent symptoms), were caused by bacterial pneumonia, and that maybe bacterial pneumonia was the primary, not the secondary, infection.
• Does pre-existing exposure to bacterial pneumonia lead to a higher propensity to acquire a viral infection, such as SARS-CoV-2? And we suggest that SARS-Cov-2 infection may mask or be secondary to pneumonia infection and not necessarily the other way around, in whichSARS-CoV-2 is assumed to lead to bacterial pneumonia as a secondary infection.
• Given this the actual burden of risk to hospitalized patients may not have been SARS-CoV-2 (or other viruses) at all but bacterial pneumonia.
• High rates of ventilator induced pneumonia are confounded by changes in protocols, delays in admission, and overuse of ventilation etc. and estimates of rates of attribution to SARS-CoV-2 cannot therefore be relied upon. Respiratory deaths in hospitals may therefore have beeb caused by bacterial pneumonia but wrongly attributed to SARS-CoV-2.
• The pattern of spread of SARS-CoV-2 in spring 2020, and the geographical concentration of the SARS-CoV-2 mortality toll is not what one would expect from a spreading respiratory virus. It is highly localised in specific geographically distant regions and cities. It is a pin-point pandemic.
• Under modern sanitary conditions large scale pneumonia outbreaks in highly concentrated areas are unlikely to occur naturally. We must look elsewhere for explanations, including the possibility of human agency.
• Given that rates of pneumonia deaths in 2020 were similar to those seen in previous years, changes in ventilation policy and practices coupled with new PCR testing, would be enough to cause the pin-point pandemic effect.
• The central question is therefore: Was SARS-CoV-2 a bystander or decoy virus and were bacterial pneumonia deaths mistakenly or intentionally used as proof that SARS-CoV-2 was a deadly respiratory pathogen?

We have investigated the pneumonia hypothesis: that a proportion of covid-19 deaths, those with associated respiratory symptoms (rather than deaths coded as covid-19 because of a positive PCR test, that are absent symptoms), were caused by bacterial pneumonia, and that bacterial pneumonia was the primary, not the secondary, infection.

We asked: does pre-existing exposure to bacterial pneumonia lead to a higher propensity to acquire a viral infection, such as influenza or SARS-CoV-2? We suggest that maybe SARS-Cov-2 infection (or at the very least a positive test for it) can be secondary to pneumonia, or it can mask pneumonia, and not necessarily the other way around.

Therefore, was SARS-CoV-2 a coincidental, bystander, infection simply present at the time of presentation or testing and the actual burden of risk to patients is not SARS-CoV-2 (or other viruses) at all but bacterial pneumonia?

We have found that high rates of ventilator induced pneumonia, attributed to SARS-CoV-2, are confounded by changes in protocols, delays in admission, overuse of ventilation and classification biases and therefore any claim that SARS-CoV-2 was uniquely dangerous cannot be relied upon. Bacterial infection is ever present at the scene.

The pattern of spread of SARS-CoV-2 in spring 2020, and the geographical concentration of the SARS-CoV-2 mortality toll is not what one would expect from a respiratory virus. It is highly localised in specific geographically distant regions and cities.

It is a pin-point pandemic.

Under modern sanitary conditions large scale pneumonia outbreaks in highly concentrated areas are unlikely to occur naturally. Changes in ventilation policy and practices coupled with new PCR testing, would be enough to cause the pin-point pandemic effect.

The central question remains:

Was SARS-CoV-2 used as a decoy and were bacterial pneumonia deaths used as proof that SARS-CoV-2 was a deadly respiratory pathogen?

Events are akin to a scene from an Agatha Christie novel where SARS-CoV-2, a bystander used as a decoy, is guilty of the crime, with ventilation as the accomplice, but the actual criminal, who has got off scot-free, is in fact bacterial pneumonia.

In other words, SARS-CoV-2 has been framed.

This is taken from a long document. Read the rest here substack.com

Header image: CareNow

Some bold emphasis added

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