Unmasking the ‘Long Covid’ Vaccine Injury Cover Story
In Part 1: Poisoned, Not Infected, we explored a paradigm-shattering idea: What if many illnesses blamed on “viruses” are in fact caused by poisons and toxins, with our bodies’ own healing responses misinterpreted as infection?
This concept builds on the “Xenogen Hypothesis,” which reframes so-called viruses as internal exosome messengers – particles released by our stressed or damaged cells to communicate distress, not to attack us[1][2].
In this view, toxic exposures – chemical pollutants, drugs, even electromagnetic stress – act as the true triggers of disease, while “viral” particles are often misidentified bystanders or effects of the toxic insult.
Fever, fatigue, cough, inflammation – these familiar “infection” symptoms are actually signs of the body detoxifying and repairing itself, not being overrun by a foreign invader[3][4].
One dramatic real-world example was the 2019 vaping lung injury outbreak known as EVALI (E-Cigarette or Vaping-Associated Lung Injury). Hundreds of young Americans were suddenly hospitalized with what looked exactly like a severe viral pneumonia: cough, chest infiltrates on X-ray, respiratory failure.
Doctors scrambled to find a pathogen, even prescribing antivirals and antibiotics[5][6]. But ultimately no virus was found – instead, investigators traced the illness to a chemical toxin: vitamin E acetate contaminating illicit vape cartridges[7].
Once the toxin was identified, it explained everything. Vitamin E acetate in the lungs triggered a severe inflammatory response that perfectly mimicked a viral infection[8].
The “contagion” was an illusion; patients fell ill around the same time simply because they shared the same toxic exposure source, creating a cluster that fooled experts into blaming an infectious outbreak[9][6].
Crucially, EVALI taught us three key lessons (as detailed in Part 1): (1) Toxic injury can produce clinical signs indistinguishable from infection; (2) When groups of people are exposed to the same toxin, their simultaneous illnesses can mimic an epidemic; and (3) the body’s response to toxins includes releasing a swarm of inflammatory exosomes – tiny vesicles loaded with “danger” signals – that can spread distress signals system-wide[10][11].
In EVALI, patients’ damaged lung cells released massive amounts of exosomes, triggering fevers, immune activation, and even damage in organs beyond the lungs[12]. These exosomes were indistinguishable from virus particles under a microscope and carried bits of genetic material and proteins – yet they were not a virus, but the body’s own toxic spillover messengers[13][14].
Let that sink in: a person poisoned by a chemical can exhibit an illness that looks infectious, even propagating harm to other cells via exosomes – with no virus involved whatsoever.
In a landmark 2018 experiment, researchers demonstrated this mechanism: Mice given a toxic overdose of acetaminophen (Tylenol) suffered severe liver damage and released “virus-sized” exosome particles into their blood.
When those exosomes were isolated and injected into healthy mice, they caused the same pattern of liver injury in the new hosts – even though the healthy mice were never exposed to the drug itself[15][16].
The exosomes from the poisoned mice carried the molecular “alarm signals” of damage and spread them to other cells, essentially transmitting an illness without an actual infectious agent[4][17].
This stunning result, published in Scientific Reports in 2018, proved that a toxin-exposed cell can export its distress to otherwise healthy cells through exosomes, inducing “infectious-like” disease features in the absence of any virus1.
In summary, Part 1 revealed a hidden truth: We are sometimes poisoned, not infected. Our bodies’ reactions to toxins – from lung-damaging vape oils to overdose of common drugs – can masquerade as contagious disease.
Exosomes, not viruses, often carry the signals of injury. Modern medicine’s fixation on germs may be blinding it to this reality, causing true causes (toxic exposures) to be overlooked while blaming “a virus” by default.
Now, in Part 2, we confront the next logical question: If toxins can so easily be mistaken for infections, could the converse also be true – that a supposed “post-viral syndrome” is hiding an ongoing toxic or iatrogenic (treatment-caused) problem?
Specifically, we turn to Long COVID: the baffling, debilitating condition affecting millions after the pandemic. We investigate the provocative possibility that “Long COVID” was seized upon – or even exaggerated in definition – to mislead people, to divert attention from the mounting adverse effects of mass vaccination and other toxic exposures in the COVID era.
Using the insights from Part 1, we’ll dissect Long COVID’s nature and ask: Are patients really suffering from a lingering virus, or are they actually poisoned (by spike proteins, immune dysregulation, and environmental co-factors) – and misdiagnosed as “infected”? The implications could not be more profound.
The Emergence of “Long COVID” – Mysterious Malady or Misdiagnosis?
By late 2020, even before vaccines were rolled out, reports began surfacing of people who had recovered from acute COVID-19 only to experience a host of persistent, strange symptoms for months on end.
This condition soon earned the name “Long COVID” (also called post-COVID syndrome or PASC). Unlike the classic recovery trajectory from viral illness, these patients weren’t getting completely better; instead, they were plagued by issues like unrelenting fatigue, brain fog, shortness of breath, chest pain, exercise intolerance, disturbed sleep, and autonomic nervous system dysfunction (for example, POTS – sudden dizziness and heart palpitations upon standing)[18][19].
Over 200 different symptoms have been linked to Long COVID across organ systems[20] – from neurological problems and cognitive impairment (“feeling in a haze”) to gastrointestinal troubles and loss of taste/smell[21].
In essence, Long COVID can manifest as a chronic multi-system illness, often resembling conditions we already knew: myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS), fibromyalgia, mast cell activation syndrome, reactive arthritis, small-fiber neuropathy, and other post-viral or autoimmune-like disorders[22][23].
Long COVID’s impact has been huge. By 2022, about 1 in 13 adults in the U.S. (7.5 percent) reported lingering symptoms long after a COVID infection – with higher rates in middle-aged adults and women[24][25].
Early studies globally produced alarming estimates (10 to 30 percent or more of all COVID cases) developing some long-term sequelae[26], though more recent analyses suggest the risk is lower in the vaccinated and with newer variants.
Still, even a conservative figure of a few percent of hundreds of millions of infections means millions of people worldwide have experienced Long COVID in some form.
Its symptoms can range from merely annoying to completely disabling, and for a subset of sufferers, daily life has been upended. Governments and health organizations have acknowledged Long COVID as a serious public health issue – albeit one defined more by patient reports than clear-cut tests.
There is no definitive diagnostic test for Long COVID; it remains a clinical syndrome, a collection of symptoms with fuzzy boundaries. By definition, it’s generally when symptoms last beyond 3 months after infection and can’t be explained by other causes[27].
Mainstream medicine, operating under the assumption that COVID-19 is purely a viral illness, has theorized that Long COVID must somehow be a consequence of the virus lingering or the immune system not resetting. Leading hypotheses include:
- Viral persistence: Fragments of SARS-CoV-2 (especially the spike protein) might remain in the body (in tissues like the gut or brain) long after the acute phase, continually provoking the immune system. For instance, researchers have found bits of viral RNA or spike protein in some patients’ blood or stool months later, fueling the idea that a “reservoir” of virus could be driving symptoms.
- Autoimmune reactions: The infection might have “broken” the immune tolerance, causing the body to attack its own cells (autoimmunity) even after the virus is gone[28]. This is supported by findings of novel autoantibodies in some Long COVID patients, and overlaps with diseases like lupus or rheumatoid arthritis.
- Latency reactivation: COVID-19 might reactivate latent viruses in the body – for example, Epstein-Barr virus (EBV) or varicella zoster – leading to chronic symptoms. Notably, EBV reactivation (the virus that causes mono) has been detected in many Long COVID cases and correlates with severity of fatigue and brain fog.
- Microclotting and endothelial damage: Some studies find tiny persistent blood clots and signs of blood vessel injury in Long COVID patients, suggesting the virus triggered a coagulation and vascular dysfunction that never fully resolved. These microclots could impair oxygen delivery to tissues, causing brain fog and exercise intolerance.
- Gut dysbiosis and reservoirs: The virus may upset the microbiome or even hide in the intestines, from where viral particles or inflammatory molecules translocate into circulation and perpetuate illness[29].
- Residual organ damage: In those who had severe COVID (e.g. on ventilators), Long COVID could simply be the long recovery from organ damage (lung scarring, nerve damage, etc.). However, puzzlingly, many Long COVID cases arose even after mild initial infections.
Mainstream researchers freely admit these are theories – “we don’t know exactly how COVID causes ongoing problems, but we have ideas”[28] – and that Long COVID likely has multiple subtypes or causes.
There is lively debate and ongoing research. But one assumption is rarely questioned: that whatever Long COVID is, it must ultimately stem from the virus (directly or indirectly). After all, by definition, the affected people had COVID-19 at some point (or at least are presumed to have).
The narrative is: “The virus did something that left these people unwell for months or years.”
Yet here is where the insights from Part 1 urge us to ask a provocative question: What if the assumption is wrong? What if SARS-CoV-2 is not the sole culprit behind Long COVID?
Could it be that some other factor – perhaps related to the pandemic but not the virus itself – is causing many of these chronic symptoms? And could the label “Long COVID” be acting as a convenient bucket to toss these sufferers into, rather than looking deeper for the true causes (which might be uncomfortable for authorities to acknowledge)?
The Overlap No One Talks About: Long COVID and Post-Vaccine Injuries
To anyone following both Long COVID and COVID vaccine side effect reports, an intriguing pattern emerged by 2021-2022: The complaints were astonishingly similar.
People receiving mRNA vaccines – especially after multiple doses – began to report problems like chronic fatigue, brain fog, dysautonomia (POTS), neuropathic pain, joint aches, headaches, even tinnitus and vision issues persisting for weeks or months after vaccination.
If that sounds familiar, it’s because it mirrors the hallmark symptoms of Long COVID. In fact, some clinicians started using a term “Long Vax” to describe this post-vaccination syndrome – anecdotally noting that if they didn’t know the patient’s history, they might just as well diagnose “Long COVID” based on the symptom picture.
Could it be that the same biological phenomenon underlies both conditions? After all, both COVID infection and the vaccines introduce a common element to the body: the coronavirus spike protein (the virus via infection; the mRNA or adenovirus vaccines via cellular production of spike antigen).
The spike protein is a known pathogenic element – it can trigger inflammation, clotting, and immune responses on its own. So it’s plausible that a lingering spike protein (or the body’s immune reaction to it) is driving much of the pathology in Long COVID and in vaccine-injury cases.
The key difference is how the spike got there – via virus or injection – but the downstream effects might be quite similar.
Recent research strongly supports this idea. In early 2025, a team at Yale led by immunologist Akiko Iwasaki published an in-depth study of what they termed “Post-Vaccination Syndrome (PVS)”, examining 42 patients who became chronically ill after COVID vaccination2.
These patients had no evidence of prior COVID-19 infection in many cases (they had negative PCR tests or antibody tests, and fell ill shortly after the shot)[30], yet they were exhibiting problems much like Long COVID.
The Yale researchers found immune system irregularities in the PVS patients: shifts in T cell populations and cytokines indicating an inflammatory state, as well as signs of latent virus reactivations (notably Epstein-Barr virus) and the presence of spike protein in the blood[31][32].
Strikingly, some of these vaccine-injured patients – even those who never had the actual virus – had measurable SARS-CoV-2 spike protein in their circulation up to >700 days (nearly two years) after vaccination[33].
In a typical case, spike antigenemia from a vaccine should vanish within a week or two; but here it was persisting for months or years, long after the mRNA or viral vector had done its job.
This echoes findings in Long COVID patients: for example, one study detected free spike protein in the plasma of Long COVID sufferers a year after infection, whereas recovered individuals had none3.
Both groups – Long COVID and Long Vax – also showed a propensity for reawakening dormant viruses (like EBV) and evidence of chronic immune activation or autoimmunity.
It’s as if the immune system, once jolted by spike (from virus or vaccine), remained stuck in overdrive or dysfunction.
These parallels did not go unnoticed by those willing to look. In fact, by 2023 some doctors began openly speculating that a significant portion of what was being labeled “Long COVID” – especially in vaccinated individuals – could actually be misdiagnosed vaccine injury.
Dr. Pierre Kory, a physician known for treating COVID and vaccine injury patients, observed that many of his long-hauler patients were not classic post-infection cases at all, but people whose symptoms started after vaccination.
In a candid interview, one such doctor claimed that “70 percent of the people I treat who were told they have long COVID are actually suffering from long-term vaccine effects” – essentially “Long Vax, not Long COVID.”4
This provocative claim went viral on social media, phrased bluntly as “Long COVID is really code for vaccine injuries.”
Public health officials and fact-checkers were quick to dismiss such statements as misinformation. They correctly pointed out that Long COVID was identified well before vaccines existed, and that numerous studies have found that vaccinated people have a lower risk of developing Long COVID than the unvaccinated (suggesting the shots protect against long-term symptoms)[34].
Indeed, it’s true that early long-hauler reports came in 2020 from unvaccinated patients. However, this is not the whole story. Crucially, the official stance that “vaccines rarely cause lasting side effects”[35] has a major blind spot: the lack of robust tracking and acknowledgement of post-vaccine syndrome.
Government safety systems were geared to catch acute adverse events (like allergic reactions, myocarditis within days, or blood clots within weeks), but not chronic, multi-system illness developing over months.
If a vaccinated person with no prior COVID infection starts suffering fatigue, brain fog, and strange nerve pains three months after their shots, will any doctor or health agency link it to the vaccine?
Or will they simply ask, “Maybe you had COVID and didn’t realize – you probably have Long COVID”? The latter scenario has been playing out repeatedly, according to patient reports.
Many such individuals (who often test negative for past infection or lack nucleocapsid antibodies, indicating no natural infection) are nonetheless told by doctors that it must be “post-COVID” causing their ills, because the vaccine is assumed “unlikely to do that.”
This convenient diagnostic labeling means the vaccine’s role stays off the radar.
This is taken from a long document. Read the rest here substack.com