COVID19: The difference between the Virus and the Disease
The Mainstream media and therefore the general public have some misconceived notions about the current pandemic often referred to as COVID-19. This document offers an explanation for the Medical Professionals, Mainstream Media, and the general public to clear up the issues surrounding the pandemic.
The Virus SARS-CoV-2 is ‘Novel’ meaning it has not been seen before and is rather an unknown quantity. There is much debate about its origin into which very technical discussions regarding its Genome and possible lineage are being investigated vigorously elsewhere.
According to the consensus Germ Theory, the Virus when encountered by Humans tries to find a location for itself a suitable place where it can multiply. In the theory all viruses are claimed to do that, they cannot replicate outside a host medium, be that a live specimen, or in a specialized petri dish.
The virus SARS-CoV-2 finds and can attach itself to the human ACE2 receptor which is present in vast numbers on the outside of cells of many human organs. This is the first oversight of the Medical profession. In Germ Theory there are many ways that any virus can enter the human body. Infectious DISEASES like Influenza are THOUGHT to be exclusively spread by aerosol contact, breathing in virus laden air expelled by an existing and infectious host.
The Disease labelled COVID-19 is a respiratory disease caused when the virus enters the human lungs (Alveolar) and lodges there to replicate. The effects are the same as with the start of an influenza infection, but take longer to progress and can and do become very serious in some people.
Now information is hard to come by, but there is anecdotal evidence of people getting digestive disorders as early symptoms, they do not express the flu like symptoms. They sometimes get those later, MUCH later!
This brings me to the recent information out of China regarding the recent outbreak in Beijing and the virus being found on the preparation boards used to process Salmon!
Now some old history, in 1948 I contracted Polio, spent 6 weeks in hospital, the local outbreak was traced to the local swimming pool, the only one for miles around and very popular, but the epicentre of the local cluster of polio sufferers. The Polio Virus was found to be in the often very wet wooden laths that made the changing room benches. Those benches were removed and replaced with metal ones there was some serious disinfections used and the outbreak wound down and stopped.
Do you see the reason for this history lesson, Wet or damp Wood can sustain viral loads for some longer time than virus particles as an aerosol, Plastic preparation boards are not much better and become criss-crossed with grooves made into their once smooth surfaces.
This then suggest rather strongly that the virus was being carried around upon the prepared food that did NOT require cooking! From a newly infectious host to the food preparation board and from there to the prepared food. It is highly probable that the infection is expelled by infected host with the COVID-19 disease.
The virus entered the new host via the mouth and lodged in the gut where it found ACE2 receptors on cells in which to reside and replicate. The Hosts auto-immune system would soon be waging a low level war against this invader and a long incubation period occurs until the virus, note it is NOT COVID-19 at this time, overcomes some cells and releases its new cohorts some of which enter the blood stream and these can then find their way into the hosts lungs and become a COVID-19 infection.
Once into the blood stream the virus can lodge in any convenient ACE2 receptor on any cell in the human body. In this way the original Virus infection may visit several organs before reaching the lungs. Thus, the original infection in the Gut can slowly spread into other organs before reaching the lungs and causing the flu like symptoms.
During the initial 14 day quarantine the virus replicates in the gut eventually spreading to secondary organs where it also takes 14 day to mature to replication state.
This is probably good news in as much that only when the virus reaches the hosts lungs and after an incubation period become infectious via aerosol’s expelled by coughs and sneezes. It is bad news regarding quantifying the true time period between original infection and being infectious.
This silent long incubation period answers many of the puzzles around SARS-COV-2 infections and tests that show positive from Asymptomatic hosts long after period of quarantine like the Essendon Footballer in Australia.
It would therefore seem that mandatory testing and repeat testing of everyone is required of those who had contact with other hosts. It also raised the question of cold foods that come in wet or damp containers and prepared outside the home.
There is then the rather large problem of where the other freshly minted Gut Virus cohort goes, out and into the sewerage system where there are many nutrients and possible hosts. A task being investigated by some.
A monumentally difficult task.
Names will be needed for diseases in hosts who have SARS-COV-2 infections in other organs as there are many that have the ACE2 receptor as part of their cell make-up.
But then again, the above may not apply for those who believe the competing theory on viruses.
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