Covid ‘Vaccines’ Could Be Considered Genetic Warfare

Written by Dr. Lewis Coleman, MD on July 24, 2024. Posted in Current News

Introduction by Richard C. Cook, Lead Investigator, American Geopolitical Institute:

It’s increasingly obvious that the COVID “plandemic” used the initial worldwide launch of the weaponized COVID-19 virus merely to get the ball rolling on the mass genocide plot that continues to devastate humanity.

But the real objective of the release of the contagion was clearly to trigger the widespread administration of the mRNA “vaccine.”

The horrors of the mRNA assault are only beginning to be understood. The “vaccine” causes the release within the human body of “spike protein” and other substances that act as long-term systemic poisons.

The main target is the human cardio-vascular system, with myocarditis appearing frequently, along with sudden death from heart failure. Even though the U.S. government gave Pfizer and other manufacturers immunity against lawsuits, the legal challenges have begun to unfold here and abroad.

“Excess deaths” from heart disease, cancer, and other causes are being documented and studied worldwide.

The COVID mass genocide is without doubt the most important public event of the last decade, perhaps of the 21^st century or even modern times. Yet it is being ignored by the mainstream media and their political favorites, including Joe Biden and Donald Trump during the run-up to November’s presidential election.

Perhaps this is not surprising, since both Biden and Trump, during their respective presidencies, presided over the “plandemic,” its generation and spread, and its devastating results.

Despite growing public awareness of what our leaders have done to the people supposedly under their care, analysis of the medical aspects of the mRNA plague are all over the map.

This is due, in part, to the lack of a unified theory of disease that explains what really happens when the human body is under the type of assault the “vaccine” causes. In particular, we lack detailed knowledge of how the insult to the cardio-vascular system proceeds from initial inoculation to heart failure and possible death.

Enter Dr. Lewis Coleman, a retired American anesthesiologist, who has spent much of his career researching and writing about stress theory and has been lately publishing articles that focus on “Long COVID,” its causes, and its possible treatment.

Following is a short biographical sketch:

Dr. Lewis S. Coleman is Chair of the Science and Education Board of the American Institute of Stress. He is a board-certified anesthesiologist who completed his BS degree in biology at the Ohio State University, obtained his MD degree from New York Medical College, and completed his surgical internship and anesthesiology residency at UCLA, followed by 40 years in private practice.

Coleman’s basic sciences instruction at NYMC miraculously coincided with the two-year sojourn of Dr. Johannes Rhodin, who was retained by the school to reform its curriculum. Dr. Rhodin was a famous researcher and expert on the stress theory of Dr. Hans Selye. His lectures devastated the dogma of classical physiology and convinced Coleman that stress theory represented the future of medicine.

Many years later, these lectures enabled Coleman to identify Selye’s long-sought Mammalian Stress Mechanism. This promises to revolutionize medicine and provide a new era of health, longevity, and freedom from the eternal scourge of disease and premature death. Coleman sets forth his ideas in his important new book, 50 Years Lost in Medical Advance: The Discovery of Hans Selye’s Stress Mechanism.

Dr. Coleman now writes as follows, starting with a discussion of heart disease.

Heart Disease and Stress Theory

This essay explains the nature of heart disease and its close associations with morbid obesity, cancer, diabetes, and hypertension in terms of stress theory.

Heart disease, as well as medicine in general, has always suffered for lack of an effective theory that explains the nature of disease and enables physicians to direct their treatments at its cause instead of guessing their effectiveness based on fickle symptoms.

I had the great good fortune to attend my basic medical science classes at New York Medical College during the last two years of the sojourn of Dr. Johannes Rhodin, an internationally famous Swedish surgeon, researcher, and expert on medical stress theory who had been retained by the school to upgrade its curriculum.

As a result, I got the benefit of his basic science curriculum that included his lectures describing stress theory, which had been the “prevailing paradigm” of international medical research for about thirty years after the discovery of DNA.

When the molecular mechanism of DNA was discovered and described by Watson and Crick in 1953, the medical experts and researchers of that time immediately realized that although DNA explains how the “genetic blueprint” is retained and replicated by every cell in the body, it fails to explain how genetic information is converted into embryological development, and it likewise fails to explain the nature of disease, blood flow regulation, organ regulation, or confer any useful treatments.

These deficiencies focused attention on the ideas of Dr. Hans Selye, who hypothesized that a single “stress mechanism” works closely with DNA to convert genetic information into embryological development, and then remains active for the duration of life to repair tissues and regulate organs.

Dr. Rhodin’s lectures on stress theory were the highlight of my medical school experience. I became an adherent of medical stress theory in the manner of a religious conversion.

Dr. Rhodin openly feared that stress theory might be forgotten unless someone in our generation—meaning our medical school class—could somehow discover a testable description of the hypothetical stress mechanism that could confirm stress theory and explain how it worked.

The search for this elusive mechanism had consumed the careers of hundreds of researchers, thousands of tortured test animals, and millions (today billions) of dollars, and was even then being abandoned.

As Dr. Rhodin feared, it is now nearly forgotten.

Little did I dream that I would ever become involved with such an esoteric theory, but that is what happened. Some thirty years after I became an anesthesiologist, I discovered fresh information about coagulation factor VIII, which I found so fascinating that I began to explore PubMed for relevant information using the Internet and advanced computer technology that hadn’t existed during my education.

The more I learned the more curious I became. Finally, after six years of intense toil, I was astonished to realize that I had discovered the long sought “mammalian stress mechanism” (MSM) that had been the subject of Dr. Rhodin’s lectures.

As he predicted, this discovery promises to revolutionize medicine, including the understanding and treatment of heart disease.

The Nature of Disease

The “mammalian stress mechanism” (MSM) functions continuously throughout life to repair tissues, regulate blood flow, and determine organ function in accord with environmental stresses.

Normally it operates efficiently and unobtrusively, but when it becomes hyperactivated by unrelenting combinations of excessive environmental stresses, it begins to consume and waste its substrates and produce excessive and defective versions of its products, which are thrombin, soluble fibrin, and insoluble fibrin.

This harmful MSM hyperactivity manifests as disease.

The MSM consists of a “tissue repair” subcomponent and a “capillary gate” subcomponent. The two subcomponents share the same enzymes and substrates, so that the activity of each subcomponent exaggerates that of the other to generate positive feedback.

This focuses stress mechanism activity to repair tissues and regulate organs, but it simultaneously produces a bewildering blizzard of constantly fluctuating symptoms and manifestations that sometimes masquerade as unique diseases that are distinctly different from one another, even though all are caused by the same mechanism.

This explains their close associations. For example, the recent COVID contagion mostly killed people suffering from pre-existing diseases, and the effects of COVID injections mimic numerous known diseases.

The MSM generates and disintegrates a complex fibrillar (string like) protein polymer called insoluble fibrin into “fibrin split products” (FSP) to regulate a submicroscopic “capillary gate mechanism” that governs microvascular blood flow resistance in accord with subconscious nervous activity called “autonomic balance.”

This explains how nervous activity governs blood flow, which determines organ function.

Normally the FSP is harmlessly re-metabolized, but MSM hyperactivity causes some of the fibrillar (string-like) FSP to undergo a “shape change” to become harmful, abnormal amyloid protein.

Unlike FSP, the amyloid protein deposits in various organs and tissues, where it induces tissue repair activity including inflammation, edema, immune activity, and collagen generation that causes harmful “sclerosis” of the afflicted tissues.

This is the underlying cause of all types of chronic illnesses, which are called “rheumatoid diseases,” and it explains the close associations of morbid obesity, diabetes, hypertension, cancer, atherosclerosis, heart attacks, and congestive heart failure.

The prototypical example of rheumatoid disease is rheumatoid arthritis, where nodules replete with amyloid protein are found in the afflicted joint tissues. Amyloid protein deposits are found in the afflicted tissues of other rheumatoid diseases as well, but they are overlooked in clinical medicine because detecting amyloid necessitates painful tissue biopsy.

The abnormal tissue repair activity induced by amyloidosis automatically activates immune activity, which causes doctors to mistakenly assume that immune activity causes these diseases, much like they mistakenly assume that harmless blood glucose elevations cause diabetes.

There is no evidence that either the immune mechanism or blood glucose causes tissue damage or toxicity.

Heart Disease

Heart disease may be regarded as a “special example” of amyloidosis that exemplifies the close relationships of hypertension, diabetes, atherosclerosis, and congestive heart failure.

Morbid obesity, smoking, and emotional stress all exaggerate subclinical stress mechanism hyperactivity, which promotes amyloidosis that accelerates the capillary deterioration that normally proceeds with age. I call this “accelerated capillary senescence.”

This increases microvascular flow resistance, which manifests as essential hypertension, and undermines the ability of cells to absorb glucose from blood, which manifests as type II diabetes. This explains the close association of diabetes and hypertension.

Conventional medical theory ignores the important role of blood turbulence. The heart initiates waves of blood turbulence that propagate throughout arterial blood vessels after each heartbeat.

These waves of blood turbulence are perceived as the pulse that follows each heartbeat. The turbulence mobilizes amyloid deposits from the inner walls of arteries to maintain arterial health. As amyloidosis destroys capillaries, it causes increasing microvascular flow resistance that alters blood turbulence.

This undermines the mobilization of amyloid deposits from the inner walls of arteries. The amyloid accumulates at the greater curvatures and bifurcations of large proximal arteries where turbulence is minimal.

The amyloid deposits induce inflammatory tissue repair activity that produces atherosclerotic lesions. The reason that atherosclerosis seldom appears in the fingers and toes is that arterial diameter declines with increasing distance from the heart.

The decreasing arterial diameter exaggerates blood turbulence, and this prevents atherosclerosis in the small arteries of fingers and toes.

Increasing microvascular flow resistance simultaneously increases cardiac work and undermines the delivery of oxygen to cardiac muscle. The resulting cardiac oxygen starvation stimulates fibroblast collagen deposition in cardiac muscle tissues.

This thickens heart walls, impairs cardiac contractility, undermines cardiac output, and causes congestive heart failure.

 An epidemic of chronic disease was brought on when tobacco corporations persuaded the American government to provide its troops with free cigarettes during World War II.

Tobacco company propaganda attributed the resulting epidemic of heart disease to excessive salt intake. When that narrative failed, they blamed cholesterol for heart disease.

Cholesterol becomes trapped in atherosclerotic lesions, but there is no evidence that it causes atherosclerosis (hardening of the arteries). Cholesterol is an essential substrate for steroid hormones that are essential for life.

The body manufactures cholesterol if dietary cholesterol is inadequate.

Orthodox medicine promotes toxic “statin” drugs that lower blood cholesterol to treat atherosclerosis, but these drugs reduce life span and are deadly. Similarly, orthodox medicine recommends toxic medications that lower blood glucose, but there is no evidence that glucose is harmful, and these medications also reduce life span.

Orthodox medicine also promotes the treatment of essential hypertension with ACE (Angiotensin- converting-enzyme) inhibitors, which beneficially open the capillary gate. This improves tissue perfusion and reduces blood pressure, but sometimes causes life-threatening airway obstruction due to tissue edema.

I first learned of this problem when I was called to the emergency room to intubate a patient with this occurring. I have no desire to repeat that experience.

Thankfully, I was successful, but I was lucky because the swollen tongue obstructed the tracheal orifice, and I was obliged to employ a crude, old-fashioned “blind” technique that is nowadays mostly forgotten since the advent of fiber optic airway devices.

Were it not for my luck that night the patient would have suffocated before my eyes.

This is taken from a long document. Read the rest here vtforeignpolicy.com

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